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col i antibody gb11022 3  (Servicebio Inc)

 
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    Servicebio Inc col i antibody gb11022 3
    Evaluation of COA-SA/CLT-siSnail micelles in treatment of UUO. (A) Representative H&E and Masson staining images of kidney tissue sections from patients with CKD. Scale bars, 50 μm. (B) Representative α -SMA and E-cadherin immunofluorescence staining images from kidney tissue sections of patients with CKD. Scale bars, 100 μm. (C) Flow diagram of treatment of different preparations against UUO-induced renal fibrosis. (D) Cr and BUN levels on Day 14 after treatment. (E) Contralateral renal index (left) and ligation side renal index (right) of UUO mice after treatment. (F) Renal tubular injury score of UUO mice following treatment. (G) The percentage of collagen area in renal tissue section by Masson staining. (H) Average optical density analysis for COL I, COL IV, FN, VIM, α -SMA, E-CAD, and SNAIL immunohistochemical staining. (I) Real-time qPCR analysis of renal mRNA levels of Acta2 , Col1a1 , Col4a1 , Cadh1 , Fn-1 , Vim , and Snai1 in sham and UUO mice on Day 14. GAPDH mRNA as internal control. Data represent mean ± SD ( n = 5). ∗ P < 0.05; ∗∗ P < 0.01; ∗∗∗ P < 0.001; ∗∗∗∗ P < 0.0001 vs . UUO; # P < 0.05; ## P < 0.01; ### P < 0.001; #### P < 0.0001 vs . sham. ns, not significant.
    Col I Antibody Gb11022 3, supplied by Servicebio Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/col+i+antibody+gb11022+3/pmc12750359-59-2-24?v=Servicebio+Inc
    Average 86 stars, based on 1 article reviews
    col i antibody gb11022 3 - by Bioz Stars, 2026-08
    86/100 stars

    Images

    1) Product Images from "Targeting renal tubular epithelial cells via a dual functionalized oligosaccharide self-assembly in the management of acute and chronic kidney diseases"

    Article Title: Targeting renal tubular epithelial cells via a dual functionalized oligosaccharide self-assembly in the management of acute and chronic kidney diseases

    Journal: Acta Pharmaceutica Sinica. B

    doi: 10.1016/j.apsb.2025.09.017

    Evaluation of COA-SA/CLT-siSnail micelles in treatment of UUO. (A) Representative H&E and Masson staining images of kidney tissue sections from patients with CKD. Scale bars, 50 μm. (B) Representative α -SMA and E-cadherin immunofluorescence staining images from kidney tissue sections of patients with CKD. Scale bars, 100 μm. (C) Flow diagram of treatment of different preparations against UUO-induced renal fibrosis. (D) Cr and BUN levels on Day 14 after treatment. (E) Contralateral renal index (left) and ligation side renal index (right) of UUO mice after treatment. (F) Renal tubular injury score of UUO mice following treatment. (G) The percentage of collagen area in renal tissue section by Masson staining. (H) Average optical density analysis for COL I, COL IV, FN, VIM, α -SMA, E-CAD, and SNAIL immunohistochemical staining. (I) Real-time qPCR analysis of renal mRNA levels of Acta2 , Col1a1 , Col4a1 , Cadh1 , Fn-1 , Vim , and Snai1 in sham and UUO mice on Day 14. GAPDH mRNA as internal control. Data represent mean ± SD ( n = 5). ∗ P < 0.05; ∗∗ P < 0.01; ∗∗∗ P < 0.001; ∗∗∗∗ P < 0.0001 vs . UUO; # P < 0.05; ## P < 0.01; ### P < 0.001; #### P < 0.0001 vs . sham. ns, not significant.
    Figure Legend Snippet: Evaluation of COA-SA/CLT-siSnail micelles in treatment of UUO. (A) Representative H&E and Masson staining images of kidney tissue sections from patients with CKD. Scale bars, 50 μm. (B) Representative α -SMA and E-cadherin immunofluorescence staining images from kidney tissue sections of patients with CKD. Scale bars, 100 μm. (C) Flow diagram of treatment of different preparations against UUO-induced renal fibrosis. (D) Cr and BUN levels on Day 14 after treatment. (E) Contralateral renal index (left) and ligation side renal index (right) of UUO mice after treatment. (F) Renal tubular injury score of UUO mice following treatment. (G) The percentage of collagen area in renal tissue section by Masson staining. (H) Average optical density analysis for COL I, COL IV, FN, VIM, α -SMA, E-CAD, and SNAIL immunohistochemical staining. (I) Real-time qPCR analysis of renal mRNA levels of Acta2 , Col1a1 , Col4a1 , Cadh1 , Fn-1 , Vim , and Snai1 in sham and UUO mice on Day 14. GAPDH mRNA as internal control. Data represent mean ± SD ( n = 5). ∗ P < 0.05; ∗∗ P < 0.01; ∗∗∗ P < 0.001; ∗∗∗∗ P < 0.0001 vs . UUO; # P < 0.05; ## P < 0.01; ### P < 0.001; #### P < 0.0001 vs . sham. ns, not significant.

    Techniques Used: Staining, Immunofluorescence, Ligation, Immunohistochemical staining, Control

    Evaluation of COA-SA/CLT-siSnail micelles in treatment of FA-CKD. (A) Flow diagram of the treatment of free CLT solution, COA-SA/CLT micelles, free siSnail solution, COA-SA/siSnail micelles, COA-SA/CLT-siSnail micelles, and COA-SA/CLT-siNC micelles against folate-induced renal fibrosis. (B) Renal index of FA-CKD mice after treatment. (C) Cr and BUN levels on Day 30 after treatment. (D) Renal tubular injury score of FA-CKD mice following treatment. (E) The percentage of collagen area in renal tissue section by Masson staining. (F) Average optical density analysis for COL I, COL IV, FN, VIM, α -SMA, E-CAD, and SNAIL immunohistochemical staining. (G) Real-time qPCR analysis of renal mRNA levels of Acta2 , Col1a1 , Col4a1 , Cadh1 , Fn-1 , Vim , and Snai1 in FA-CKD mice on Day 30 after treatment. GAPDH mRNA as internal control. Data represent mean ± SD ( n = 5). ∗ P < 0.05; ∗∗ P < 0.01; ∗∗∗ P < 0.001; ∗∗∗∗ P < 0.0001 vs . FA-CKD; # P < 0.05; ## P < 0.01 vs . vehicle. ns, not significant.
    Figure Legend Snippet: Evaluation of COA-SA/CLT-siSnail micelles in treatment of FA-CKD. (A) Flow diagram of the treatment of free CLT solution, COA-SA/CLT micelles, free siSnail solution, COA-SA/siSnail micelles, COA-SA/CLT-siSnail micelles, and COA-SA/CLT-siNC micelles against folate-induced renal fibrosis. (B) Renal index of FA-CKD mice after treatment. (C) Cr and BUN levels on Day 30 after treatment. (D) Renal tubular injury score of FA-CKD mice following treatment. (E) The percentage of collagen area in renal tissue section by Masson staining. (F) Average optical density analysis for COL I, COL IV, FN, VIM, α -SMA, E-CAD, and SNAIL immunohistochemical staining. (G) Real-time qPCR analysis of renal mRNA levels of Acta2 , Col1a1 , Col4a1 , Cadh1 , Fn-1 , Vim , and Snai1 in FA-CKD mice on Day 30 after treatment. GAPDH mRNA as internal control. Data represent mean ± SD ( n = 5). ∗ P < 0.05; ∗∗ P < 0.01; ∗∗∗ P < 0.001; ∗∗∗∗ P < 0.0001 vs . FA-CKD; # P < 0.05; ## P < 0.01 vs . vehicle. ns, not significant.

    Techniques Used: Staining, Immunohistochemical staining, Control



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    Evaluation of COA-SA/CLT-siSnail micelles in treatment of UUO. (A) Representative H&E and Masson staining images of kidney tissue sections from patients with CKD. Scale bars, 50 μm. (B) Representative α -SMA and E-cadherin immunofluorescence staining images from kidney tissue sections of patients with CKD. Scale bars, 100 μm. (C) Flow diagram of treatment of different preparations against UUO-induced renal fibrosis. (D) Cr and BUN levels on Day 14 after treatment. (E) Contralateral renal index (left) and ligation side renal index (right) of UUO mice after treatment. (F) Renal tubular injury score of UUO mice following treatment. (G) The percentage of collagen area in renal tissue section by Masson staining. (H) Average optical density analysis for COL I, COL IV, FN, VIM, α -SMA, E-CAD, and SNAIL immunohistochemical staining. (I) Real-time qPCR analysis of renal mRNA levels of Acta2 , Col1a1 , Col4a1 , Cadh1 , Fn-1 , Vim , and Snai1 in sham and UUO mice on Day 14. GAPDH mRNA as internal control. Data represent mean ± SD ( n = 5). ∗ P < 0.05; ∗∗ P < 0.01; ∗∗∗ P < 0.001; ∗∗∗∗ P < 0.0001 vs . UUO; # P < 0.05; ## P < 0.01; ### P < 0.001; #### P < 0.0001 vs . sham. ns, not significant.
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    Evaluation of COA-SA/CLT-siSnail micelles in treatment of UUO. (A) Representative H&E and Masson staining images of kidney tissue sections from patients with CKD. Scale bars, 50 μm. (B) Representative α -SMA and E-cadherin immunofluorescence staining images from kidney tissue sections of patients with CKD. Scale bars, 100 μm. (C) Flow diagram of treatment of different preparations against UUO-induced renal fibrosis. (D) Cr and BUN levels on Day 14 after treatment. (E) Contralateral renal index (left) and ligation side renal index (right) of UUO mice after treatment. (F) Renal tubular injury score of UUO mice following treatment. (G) The percentage of collagen area in renal tissue section by Masson staining. (H) Average optical density analysis for COL I, COL IV, FN, VIM, α -SMA, E-CAD, and SNAIL immunohistochemical staining. (I) Real-time qPCR analysis of renal mRNA levels of Acta2 , Col1a1 , Col4a1 , Cadh1 , Fn-1 , Vim , and Snai1 in sham and UUO mice on Day 14. GAPDH mRNA as internal control. Data represent mean ± SD ( n = 5). ∗ P < 0.05; ∗∗ P < 0.01; ∗∗∗ P < 0.001; ∗∗∗∗ P < 0.0001 vs . UUO; # P < 0.05; ## P < 0.01; ### P < 0.001; #### P < 0.0001 vs . sham. ns, not significant.
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    Image Search Results


    Evaluation of COA-SA/CLT-siSnail micelles in treatment of UUO. (A) Representative H&E and Masson staining images of kidney tissue sections from patients with CKD. Scale bars, 50 μm. (B) Representative α -SMA and E-cadherin immunofluorescence staining images from kidney tissue sections of patients with CKD. Scale bars, 100 μm. (C) Flow diagram of treatment of different preparations against UUO-induced renal fibrosis. (D) Cr and BUN levels on Day 14 after treatment. (E) Contralateral renal index (left) and ligation side renal index (right) of UUO mice after treatment. (F) Renal tubular injury score of UUO mice following treatment. (G) The percentage of collagen area in renal tissue section by Masson staining. (H) Average optical density analysis for COL I, COL IV, FN, VIM, α -SMA, E-CAD, and SNAIL immunohistochemical staining. (I) Real-time qPCR analysis of renal mRNA levels of Acta2 , Col1a1 , Col4a1 , Cadh1 , Fn-1 , Vim , and Snai1 in sham and UUO mice on Day 14. GAPDH mRNA as internal control. Data represent mean ± SD ( n = 5). ∗ P < 0.05; ∗∗ P < 0.01; ∗∗∗ P < 0.001; ∗∗∗∗ P < 0.0001 vs . UUO; # P < 0.05; ## P < 0.01; ### P < 0.001; #### P < 0.0001 vs . sham. ns, not significant.

    Journal: Acta Pharmaceutica Sinica. B

    Article Title: Targeting renal tubular epithelial cells via a dual functionalized oligosaccharide self-assembly in the management of acute and chronic kidney diseases

    doi: 10.1016/j.apsb.2025.09.017

    Figure Lengend Snippet: Evaluation of COA-SA/CLT-siSnail micelles in treatment of UUO. (A) Representative H&E and Masson staining images of kidney tissue sections from patients with CKD. Scale bars, 50 μm. (B) Representative α -SMA and E-cadherin immunofluorescence staining images from kidney tissue sections of patients with CKD. Scale bars, 100 μm. (C) Flow diagram of treatment of different preparations against UUO-induced renal fibrosis. (D) Cr and BUN levels on Day 14 after treatment. (E) Contralateral renal index (left) and ligation side renal index (right) of UUO mice after treatment. (F) Renal tubular injury score of UUO mice following treatment. (G) The percentage of collagen area in renal tissue section by Masson staining. (H) Average optical density analysis for COL I, COL IV, FN, VIM, α -SMA, E-CAD, and SNAIL immunohistochemical staining. (I) Real-time qPCR analysis of renal mRNA levels of Acta2 , Col1a1 , Col4a1 , Cadh1 , Fn-1 , Vim , and Snai1 in sham and UUO mice on Day 14. GAPDH mRNA as internal control. Data represent mean ± SD ( n = 5). ∗ P < 0.05; ∗∗ P < 0.01; ∗∗∗ P < 0.001; ∗∗∗∗ P < 0.0001 vs . UUO; # P < 0.05; ## P < 0.01; ### P < 0.001; #### P < 0.0001 vs . sham. ns, not significant.

    Article Snippet: Anti-collagen I (COL I) antibody (GB11022-3), anti- α -SMA antibody (GB13044), anti-Vimentin antibody (GB11192), and anti-Fibronectin (FN) antibody ( GB114057 ) were purchased from Servicebio (Wuhan, China).

    Techniques: Staining, Immunofluorescence, Ligation, Immunohistochemical staining, Control

    Evaluation of COA-SA/CLT-siSnail micelles in treatment of FA-CKD. (A) Flow diagram of the treatment of free CLT solution, COA-SA/CLT micelles, free siSnail solution, COA-SA/siSnail micelles, COA-SA/CLT-siSnail micelles, and COA-SA/CLT-siNC micelles against folate-induced renal fibrosis. (B) Renal index of FA-CKD mice after treatment. (C) Cr and BUN levels on Day 30 after treatment. (D) Renal tubular injury score of FA-CKD mice following treatment. (E) The percentage of collagen area in renal tissue section by Masson staining. (F) Average optical density analysis for COL I, COL IV, FN, VIM, α -SMA, E-CAD, and SNAIL immunohistochemical staining. (G) Real-time qPCR analysis of renal mRNA levels of Acta2 , Col1a1 , Col4a1 , Cadh1 , Fn-1 , Vim , and Snai1 in FA-CKD mice on Day 30 after treatment. GAPDH mRNA as internal control. Data represent mean ± SD ( n = 5). ∗ P < 0.05; ∗∗ P < 0.01; ∗∗∗ P < 0.001; ∗∗∗∗ P < 0.0001 vs . FA-CKD; # P < 0.05; ## P < 0.01 vs . vehicle. ns, not significant.

    Journal: Acta Pharmaceutica Sinica. B

    Article Title: Targeting renal tubular epithelial cells via a dual functionalized oligosaccharide self-assembly in the management of acute and chronic kidney diseases

    doi: 10.1016/j.apsb.2025.09.017

    Figure Lengend Snippet: Evaluation of COA-SA/CLT-siSnail micelles in treatment of FA-CKD. (A) Flow diagram of the treatment of free CLT solution, COA-SA/CLT micelles, free siSnail solution, COA-SA/siSnail micelles, COA-SA/CLT-siSnail micelles, and COA-SA/CLT-siNC micelles against folate-induced renal fibrosis. (B) Renal index of FA-CKD mice after treatment. (C) Cr and BUN levels on Day 30 after treatment. (D) Renal tubular injury score of FA-CKD mice following treatment. (E) The percentage of collagen area in renal tissue section by Masson staining. (F) Average optical density analysis for COL I, COL IV, FN, VIM, α -SMA, E-CAD, and SNAIL immunohistochemical staining. (G) Real-time qPCR analysis of renal mRNA levels of Acta2 , Col1a1 , Col4a1 , Cadh1 , Fn-1 , Vim , and Snai1 in FA-CKD mice on Day 30 after treatment. GAPDH mRNA as internal control. Data represent mean ± SD ( n = 5). ∗ P < 0.05; ∗∗ P < 0.01; ∗∗∗ P < 0.001; ∗∗∗∗ P < 0.0001 vs . FA-CKD; # P < 0.05; ## P < 0.01 vs . vehicle. ns, not significant.

    Article Snippet: Anti-collagen I (COL I) antibody (GB11022-3), anti- α -SMA antibody (GB13044), anti-Vimentin antibody (GB11192), and anti-Fibronectin (FN) antibody ( GB114057 ) were purchased from Servicebio (Wuhan, China).

    Techniques: Staining, Immunohistochemical staining, Control